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Mitochondrial reactive oxygen species (ROS) inhibition ameliorates palmitate-induced INS-1 beta cell death

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Luteolin decreases the levels of proinflammatory cytokines and increases the levels of brain-derived neurotrophic factor (BDNF) and its downstream phosphorylated mechanistic target of rapamycin (p-mTOR), synaptic proteins synaptophysin (SYN), and PSD-95, thereby affecting synaptic function and morphology