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It was found that m6A modification-associated protein (IGF2BP3), which is highly expressed in human cervical cancer, can enhance Gln metabolism, promote lactate production and secretion, and affect Treg cell differentiation by up-regulating GLS and GLUD1, thus leading to immune escape [96]

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Supplemental studies have shed light on the upstream mechanism of this signaling pathway, demonstrating that sUA provokes IR in HUA by stimulating the Nuclear factor erythroid 2-related factor 2 (Nrf2)/Heme Oxygenase 1(HO-1) signaling pathway and upregulating the regulatory thioredoxin interacting protein (TXNIP), thereby inhibiting the IRS2/PI3K/AKT signaling pathway in macrophages (57)