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Morphologically, ferroptosis is characterized by necrotic-like features, including loss of mitochondrial cristae, increased membrane density, outer membrane rupture, and reduced mitochondrial volume, while preserving plasma membrane integrity during early stages and maintaining normal nuclear morphology

The key feature of ferroptosis is the accumulation of lipid hydroperoxide, resulting from a decrease in glutathione peroxidases (GPXs)

The tissue becomes thickened, fibrotic, and less elastic

In Phase III, products of phases I and/or II are excreted from cells by multidrug resistance proteins and other ABC transporters [37]

GPCRs directly interact with heterotrimeric G proteins on the cytosolic surface of the membrane