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While OCT1 is predominantly hepatic, the high expression of OCT3 in the myocardium facilitates tissue penetration and accumulation in cardiomyocytes (Solbach et al., 2011)

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We estimated the extent of mitochondrial damage in these cells via transmission electron microscopy (TEM) (Fig

To understand mediators of cagrilintide action, we generated a transcriptomics atlas of over 530,000 cells comprising 80 neuronal cell populations across rat, mouse and macaque caudal brainstem, with spatial profiling to map distribution in the rat dorsal vagal complex (DVC)