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Neurogenic inflammation in acute migraine attacks is characterized by a TVS activation-induced release of neuropeptides, such as CGRP and substance P, from trigeminal nerve fibers at the dura, which causes increased vascular permeability, plasma protein extravasation, leukocyte infiltration, vasodilation, mast cell degranulation, activation of meningeal nociceptors, and the subsequent release of inflammatory mediators (5)

H., Ahn, J

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While these data support the notion that high glucose contributes to reduced BBB integrity, at least in part through endothelial injury, there are conflicting reports on the endothelial barrier or TJ protein expression in the context of hyperglycemia