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A deeper mechanistic understanding of how systemic signals modulate ferroptosis-immune crosstalk in vivo may enable the development of therapeutic strategies that precisely recalibrate necroinflammatory pathways, thereby delineating how targeted modulation of necroinflammatory pathways can be leveraged to uncouple cell death from adverse immune effects and thereby refine future therapeutic strategies

10.1016/j.fct.2016.07.008 [DOI] [PubMed] [Google Scholar] Pastoriza, S

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Bipolar disorder is also characterized by mutations to mtDNA and mitochondrial dysfunction caused predominantly by excess ROS (Andreazza et al

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