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Overactivation of PI3K/AKT induces premature activation of the dormant follicle pool and follicular burnout, culminating in fast depletion and eventual POF, whereas inhibition of PI3K/AKT may transpire in developing follicles, resulting in GC death and follicular atresia (Zhang et al

Taken together, these results support a model in which metformin suppresses TXNIP expression not through AMPK activation, but via mitochondrial complex I inhibition, leading to increased NADPH production, enhanced antioxidant gene expression and ROS reduction

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