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Unlike rHN, rat MOTS-c is exclusively mitochondrial, while in humans, translation may occur in the cytoplasm due to codon differences

Improves Insulin Sensitivity MOTS-C significantly improves how your cells respond to insulin, which: Lowers blood sugar levels Reduces insulin requirements Decreases fat storage Prevents or reverses type 2 diabetes Reduces cardiovascular disease risk In research studies, MOTS-C treatment reversed high-fat diet-induced insulin resistance and improved glucose metabolism even in already diabetic animals

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