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Its proposed mechanism is unusual: Selank appears to modulate the expression of GABA-A receptor genes without binding the benzodiazepine site , which is offered as an explanation for anxiolytic activity that comes without sedation, tolerance, dependence or withdrawal [3]

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In addition to its potential on the intercellular matrix, GHK-Cu may also have antioxidant and anti-inflammatory characteristics that may protect cells from damage caused by free radicals

Patients with chronic infections (hepatitis C infection

Ethics statement The animal study was approved by Oujiang Laboratory Animal Ethics Committee